A decrease in thrombocyte count was observed in (NZW x BXSB)F1 (W/B F1) mice at the age of greater than 5 mo, whereas megakaryocyte counts were found to increase in such mice. FACS analyses revealed the presence of both platelet-associated antibodies (PAA) and circulating antiplatelet antibodies. There is a correlation between the presence of these antibodies and the degree of thrombocytopenia. The transplantation of normal bone marrow cells from BALB/c nu/nu mice to W/B F1 mice was found to have preventative and curative effects on thrombocytopenia; the mice showed normal platelet counts and no evidence of circulating antiplatelet antibodies. These results indicate that thrombocytopenia in W/B F1 mice is due to the presence of antibodies to platelets. We therefore think that W/B F1 mice serve as a useful animal model of idiopathic thrombocytopenic purpura (ITP) not only for elucidating the mechanism of the development of antiplatelet antibodies, but also for characterizing autoantibodies to platelets.
Skip Nav Destination
Article navigation
Article|
June 01 1988
(NZW x BXSB)F1 mouse. A new animal model of idiopathic thrombocytopenic purpura.
N Oyaizu,
N Oyaizu
Department of Pathology, Kansai Medical University, Osaka, Japan.
Search for other works by this author on:
R Yasumizu,
R Yasumizu
Department of Pathology, Kansai Medical University, Osaka, Japan.
Search for other works by this author on:
M Miyama-Inaba,
M Miyama-Inaba
Department of Pathology, Kansai Medical University, Osaka, Japan.
Search for other works by this author on:
S Nomura,
S Nomura
Department of Pathology, Kansai Medical University, Osaka, Japan.
Search for other works by this author on:
H Yoshida,
H Yoshida
Department of Pathology, Kansai Medical University, Osaka, Japan.
Search for other works by this author on:
S Miyawaki,
S Miyawaki
Department of Pathology, Kansai Medical University, Osaka, Japan.
Search for other works by this author on:
Y Shibata,
Y Shibata
Department of Pathology, Kansai Medical University, Osaka, Japan.
Search for other works by this author on:
S Mitsuoka,
S Mitsuoka
Department of Pathology, Kansai Medical University, Osaka, Japan.
Search for other works by this author on:
K Yasunaga,
K Yasunaga
Department of Pathology, Kansai Medical University, Osaka, Japan.
Search for other works by this author on:
S Morii
S Morii
Department of Pathology, Kansai Medical University, Osaka, Japan.
Search for other works by this author on:
N Oyaizu
Department of Pathology, Kansai Medical University, Osaka, Japan.
R Yasumizu
Department of Pathology, Kansai Medical University, Osaka, Japan.
M Miyama-Inaba
Department of Pathology, Kansai Medical University, Osaka, Japan.
S Nomura
Department of Pathology, Kansai Medical University, Osaka, Japan.
H Yoshida
Department of Pathology, Kansai Medical University, Osaka, Japan.
S Miyawaki
Department of Pathology, Kansai Medical University, Osaka, Japan.
Y Shibata
Department of Pathology, Kansai Medical University, Osaka, Japan.
S Mitsuoka
Department of Pathology, Kansai Medical University, Osaka, Japan.
K Yasunaga
Department of Pathology, Kansai Medical University, Osaka, Japan.
S Morii
Department of Pathology, Kansai Medical University, Osaka, Japan.
Online ISSN: 1540-9538
Print ISSN: 0022-1007
J Exp Med (1988) 167 (6): 2017–2022.
Citation
N Oyaizu, R Yasumizu, M Miyama-Inaba, S Nomura, H Yoshida, S Miyawaki, Y Shibata, S Mitsuoka, K Yasunaga, S Morii; (NZW x BXSB)F1 mouse. A new animal model of idiopathic thrombocytopenic purpura.. J Exp Med 1 June 1988; 167 (6): 2017–2022. doi: https://doi.org/10.1084/jem.167.6.2017
Download citation file:
Suggested Content
Email alerts
Advertisement