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T cell development in B cell-deficient mice. V. Stopping anti-mu treatment results in Igh-restricted expansion of the T suppressor cell repertoire concomitant with the development of normal immunoglobulin levels.

K T Hayglass, B Benacerraf, M S Sy

J Exp Med (1986) 164 (1): 36–49.

DOI: https://doi.org/10.1084/jem.164.1.36

Published: 01 July 1986

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Journal Articles

TAP, a novel T cell-activating protein involved in the stimulation of MHC-restricted T lymphocytes.

K L Rock, E T Yeh, C F Gramm, S I Haber, H Reiser, B Benacerraf

J Exp Med (1986) 163 (2): 315–333.

DOI: https://doi.org/10.1084/jem.163.2.315

Published: 01 February 1986

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T cell development in B cell-deficient mice. II. Serological characterization of suppressor T cell factors (TsF1) produced in normal mice and in mice treated chronically with rabbit anti-mouse IgM antibodies.

M S Sy, K T Hayglass, B Benacerraf

J Exp Med (1985) 161 (6): 1402–1413.

DOI: https://doi.org/10.1084/jem.161.6.1402

Published: 01 June 1985

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Henry G. Kunkel 1916-1983. An appreciation of the man and his scientific contributions & a bibliography of his research papers.

A G Bearn, F J Dixon, B Benacerraf

J Exp Med (1985) 161 (5): 869–895.

DOI: https://doi.org/10.1084/jem.161.5.869

Published: 01 May 1985

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Inhibition of antigen-specific T lymphocyte activation by structurally related Ir gene-controlled polymers. II. Competitive inhibition of I-E-restricted, antigen-specific T cell responses.

K L Rock, B Benacerraf

J Exp Med (1984) 160 (6): 1864–1879.

DOI: https://doi.org/10.1084/jem.160.6.1864

Published: 01 December 1984

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Antigen presentation by hapten-specific B lymphocytes. I. Role of surface immunoglobulin receptors.

K L Rock, B Benacerraf, A K Abbas

J Exp Med (1984) 160 (4): 1102–1113.

DOI: https://doi.org/10.1084/jem.160.4.1102

Published: 01 October 1984

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Analysis of thymocyte MHC specificity with thymocyte hybridomas.

E T Yeh, B Benacerraf, K L Rock

J Exp Med (1984) 160 (3): 799–813.

DOI: https://doi.org/10.1084/jem.160.3.799

Published: 01 September 1984

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Selective modification of a private I-A allo-stimulating determinant(s) upon association of antigen with an antigen-presenting cell.

K L Rock, B Benacerraf

J Exp Med (1984) 159 (4): 1238–1252.

DOI: https://doi.org/10.1084/jem.159.4.1238

Published: 01 April 1984

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Journal Articles

An antigen-specific signal is required for the activation of second-order suppressor T cells in the regulation of delayed-type hypersensitivity to 2,4,6-trinitrobenzene sulfonic acid.

M Tsurufuji, B Benacerraf, M S Sy

J Exp Med (1983) 158 (3): 932–945.

DOI: https://doi.org/10.1084/jem.158.3.932

Published: 01 September 1983

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Inhibition of antigen-specific T lymphocyte activation by structurally related Ir gene-controlled polymers. Evidence of specific competition for accessory cell antigen presentation.

K L Rock, B Benacerraf

J Exp Med (1983) 157 (5): 1618–1634.

DOI: https://doi.org/10.1084/jem.157.5.1618

Published: 01 May 1983

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The role of Ia molecules in the activation of T lymphocytes. III. Antigen-specific, Ia-restricted, interleukin 2-producing T cell hybridomas with detectable affinity for the restricting I-A molecule.

K L Rock, B Benacerraf

J Exp Med (1983) 157 (1): 359–364.

DOI: https://doi.org/10.1084/jem.157.1.359

Published: 01 January 1983

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Identification of an I-J+ antigen-presenting cell required for third order suppressor cell activation.

A Lowy, A Tominaga, J A Drebin, M Takaoki, B Benacerraf, M I Greene

J Exp Med (1983) 157 (1): 353–358.

DOI: https://doi.org/10.1084/jem.157.1.353

Published: 01 January 1983

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Journal Articles

I-J-restricted interactions in the generation of azobenzenearsonate-specific suppressor T cells.

M Takaoki, M S Sy, A Tominaga, A Lowy, M Tsurufuji, R Finberg, B Benacerraf, M I Greene

J Exp Med (1982) 156 (5): 1325–1334.

DOI: https://doi.org/10.1084/jem.156.5.1325

Published: 01 November 1982

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Two distinct mechanisms regulate the in vivo generation of cytotoxic T cells.

M S Sy, S H Lee, M Tsurufuji, K L Rock, B Benacerraf, R Finberg

J Exp Med (1982) 156 (3): 918–923.

DOI: https://doi.org/10.1084/jem.156.3.918

Published: 01 September 1982

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Analysis of T cell hybridomas. I. Characterization of H-2 and Igh-restricted monoclonal suppressor factors.

M Minami, K Okuda, S Furusawa, B Benacerraf, M E Dorf

J Exp Med (1981) 154 (5): 1390–1402.

DOI: https://doi.org/10.1084/jem.154.5.1390

Published: 01 November 1981

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Antigen- and receptor-driven regulatory mechanisms. VIII. Suppression of idiotype-negative, p-azobenzenearsonate-specific T cells results from the interaction of an anti-idiotypic second-order T suppressor cell with a cross-reactive-idiotype-positive, p-azobenzenearsonate-primed T cell target.

M S Sy, A Nisonoff, R N Germain, B Benacerraf, M I Greene

J Exp Med (1981) 153 (6): 1415–1425.

DOI: https://doi.org/10.1084/jem.153.6.1415

Published: 01 June 1981

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Hapten-specific T cell responses to 4-hydroxy-3-nitrophenyl acetyl. VIII. Suppressor cell pathways in cutaneous sensitivity responses.

M E Sunday, B Benacerraf, M E Dorf

J Exp Med (1981) 153 (4): 811–822.

DOI: https://doi.org/10.1084/jem.153.4.811

Published: 01 April 1981

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Hapten-specific T cell responses to 4-hydroxy-3-nitrophenyl acetyl. VII. Idiotype-specific suppression of plaque-forming cell responses.

D H Sherr, S T Ju, J Z Weinberger, B Benacerraf, M E Dorf

J Exp Med (1981) 153 (3): 640–652.

DOI: https://doi.org/10.1084/jem.153.3.640

Published: 01 March 1981

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Mechanisms of regulation of cell-mediated immunity. VII. Suppressor T cells induced by suboptimal doses of antigen plus an I-J-specific allogeneic effect.

J S Bromberg, B Benacerraf, M I Greene

J Exp Med (1981) 153 (2): 437–449.

DOI: https://doi.org/10.1084/jem.153.2.437

Published: 01 February 1981

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Journal Articles

Antigen- and receptor-driven regulatory mechanisms. VII. H-2-restricted anti-idiotypic suppressor factor from efferent suppressor T cells.

M H Dietz, M S Sy, B Benacerraf, A Nisonoff, M I Greene, R N Germain

J Exp Med (1981) 153 (2): 450–463.

DOI: https://doi.org/10.1084/jem.153.2.450

Published: 01 February 1981

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2 3 4

NARROW

T cell development in B cell-deficient mice. V. Stopping anti-mu treatment results in Igh-restricted expansion of the T suppressor cell repertoire concomitant with the development of normal immunoglobulin levels.

TAP, a novel T cell-activating protein involved in the stimulation of MHC-restricted T lymphocytes.

T cell development in B cell-deficient mice. II. Serological characterization of suppressor T cell factors (TsF1) produced in normal mice and in mice treated chronically with rabbit anti-mouse IgM antibodies.

Henry G. Kunkel 1916-1983. An appreciation of the man and his scientific contributions & a bibliography of his research papers.

Inhibition of antigen-specific T lymphocyte activation by structurally related Ir gene-controlled polymers. II. Competitive inhibition of I-E-restricted, antigen-specific T cell responses.

Antigen presentation by hapten-specific B lymphocytes. I. Role of surface immunoglobulin receptors.

Analysis of thymocyte MHC specificity with thymocyte hybridomas.

Selective modification of a private I-A allo-stimulating determinant(s) upon association of antigen with an antigen-presenting cell.

An antigen-specific signal is required for the activation of second-order suppressor T cells in the regulation of delayed-type hypersensitivity to 2,4,6-trinitrobenzene sulfonic acid.

Inhibition of antigen-specific T lymphocyte activation by structurally related Ir gene-controlled polymers. Evidence of specific competition for accessory cell antigen presentation.

The role of Ia molecules in the activation of T lymphocytes. III. Antigen-specific, Ia-restricted, interleukin 2-producing T cell hybridomas with detectable affinity for the restricting I-A molecule.

Identification of an I-J+ antigen-presenting cell required for third order suppressor cell activation.

I-J-restricted interactions in the generation of azobenzenearsonate-specific suppressor T cells.

Two distinct mechanisms regulate the in vivo generation of cytotoxic T cells.

Analysis of T cell hybridomas. I. Characterization of H-2 and Igh-restricted monoclonal suppressor factors.

Antigen- and receptor-driven regulatory mechanisms. VIII. Suppression of idiotype-negative, p-azobenzenearsonate-specific T cells results from the interaction of an anti-idiotypic second-order T suppressor cell with a cross-reactive-idiotype-positive, p-azobenzenearsonate-primed T cell target.

Hapten-specific T cell responses to 4-hydroxy-3-nitrophenyl acetyl. VIII. Suppressor cell pathways in cutaneous sensitivity responses.

Hapten-specific T cell responses to 4-hydroxy-3-nitrophenyl acetyl. VII. Idiotype-specific suppression of plaque-forming cell responses.

Mechanisms of regulation of cell-mediated immunity. VII. Suppressor T cells induced by suboptimal doses of antigen plus an I-J-specific allogeneic effect.

Antigen- and receptor-driven regulatory mechanisms. VII. H-2-restricted anti-idiotypic suppressor factor from efferent suppressor T cells.

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