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T Cell Receptor Complementarity Determining Region 3 Length Analysis Reveals the Absence of a Characteristic Public T Cell Repertoire in Neonatal Tolerance: The Response in the “Tolerant” Mouse within the Residual Repertoire Is Quantitatively Similar but Qualitatively Different

Emanual Maverakis, Jonathan T. Beech, Stephen S. Wilson, Anthony Quinn, Brian Pedersen, Eli E. Sercarz

J Exp Med (2000) 191 (4): 695–702.

DOI: https://doi.org/10.1084/jem.191.4.695

Published: 21 February 2000

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Journal Articles

Diversification of T Cell Responses to Carboxy-terminal Determinants within the 65-kD Heat-shock Protein Is Involved in Regulation of Autoimmune Arthritis

Kamal D. Moudgil, Tammy T. Chang, Herbert Eradat, Audrey M. Chen, Radhey S. Gupta, Ernest Brahn, Eli E. Sercarz

J Exp Med (1997) 185 (7): 1307–1316.

DOI: https://doi.org/10.1084/jem.185.7.1307

Published: 07 April 1997

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Journal Articles

Inactivation of T cell receptor peptide-specific CD4 regulatory T cells induces chronic experimental autoimmune encephalomyelitis (EAE).

V Kumar, K Stellrecht, E Sercarz

J Exp Med (1996) 184 (5): 1609–1617.

DOI: https://doi.org/10.1084/jem.184.5.1609

Published: 01 November 1996

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Journal Articles

A truncated T cell receptor repertoire reveals underlying immunogenicity of an antigenic determinant.

N K Nanda, E Sercarz

J Exp Med (1996) 184 (3): 1037–1043.

DOI: https://doi.org/10.1084/jem.184.3.1037

Published: 01 September 1996

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Journal Articles

Neonatal peptide exposure can prime T cells and, upon subsequent immunization, induce their immune deviation: implications for antibody vs. T cell-mediated autoimmunity.

R R Singh, B H Hahn, E E Sercarz

J Exp Med (1996) 183 (4): 1613–1621.

DOI: https://doi.org/10.1084/jem.183.4.1613

Published: 01 April 1996

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Journal Articles

Unresponsiveness to a self-peptide of mouse lysozyme owing to hindrance of T cell receptor-major histocompatibility complex/peptide interaction caused by flanking epitopic residues.

K D Moudgil, I S Grewal, P E Jensen, E E Sercarz

J Exp Med (1996) 183 (2): 535–546.

DOI: https://doi.org/10.1084/jem.183.2.535

Published: 01 February 1996

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Journal Articles

Recognition of multiple peptide cores by a single T cell receptor.

N K Nanda, K K Arzoo, H M Geysen, A Sette, E E Sercarz

J Exp Med (1995) 182 (2): 531–539.

DOI: https://doi.org/10.1084/jem.182.2.531

Published: 01 August 1995

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Journal Articles

T cell determinants from autoantibodies to DNA can upregulate autoimmunity in murine systemic lupus erythematosus.

R R Singh, V Kumar, F M Ebling, S Southwood, A Sette, E E Sercarz, B H Hahn

J Exp Med (1995) 181 (6): 2017–2027.

DOI: https://doi.org/10.1084/jem.181.6.2017

Published: 01 June 1995

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Journal Articles

Holes in the T cell repertoire to myelin basic protein owing to the absence of the D beta 2-J beta 2 gene cluster: implications for T cell receptor recognition and autoimmunity.

V Kumar, E Sercarz

J Exp Med (1994) 179 (5): 1637–1643.

DOI: https://doi.org/10.1084/jem.179.5.1637

Published: 01 May 1994

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Journal Articles

Dominant determinants in hen eggwhite lysozyme correspond to the cryptic determinants within its self-homologue, mouse lysozyme: implications in shaping of the T cell repertoire and autoimmunity.

K D Moudgil, E E Sercarz

J Exp Med (1993) 178 (6): 2131–2138.

DOI: https://doi.org/10.1084/jem.178.6.2131

Published: 01 December 1993

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Journal Articles

Determinant capture as a possible mechanism of protection afforded by major histocompatibility complex class II molecules in autoimmune disease.

H Deng, R Apple, M Clare-Salzler, S Trembleau, D Mathis, L Adorini, E Sercarz

J Exp Med (1993) 178 (5): 1675–1680.

DOI: https://doi.org/10.1084/jem.178.5.1675

Published: 01 November 1993

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Journal Articles

The involvement of T cell receptor peptide-specific regulatory CD4+ T cells in recovery from antigen-induced autoimmune disease.

V Kumar, E E Sercarz

J Exp Med (1993) 178 (3): 909–916.

DOI: https://doi.org/10.1084/jem.178.3.909

Published: 01 September 1993

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Journal Articles

In a small multideterminant peptide, each determinant is recognized by a different V beta gene segment.

N K Nanda, K K Arzoo, E E Sercarz

J Exp Med (1992) 176 (1): 297–302.

DOI: https://doi.org/10.1084/jem.176.1.297

Published: 01 July 1992

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Journal Articles

Donor major histocompatibility complex (MHC) peptides are presented by recipient MHC molecules during graft rejection.

G Benichou, P A Takizawa, C A Olson, M McMillan, E E Sercarz

J Exp Med (1992) 175 (1): 305–308.

DOI: https://doi.org/10.1084/jem.175.1.305

Published: 01 January 1992

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Journal Articles

T cell determinant structure: cores and determinant envelopes in three mouse major histocompatibility complex haplotypes.

G Gammon, H M Geysen, R J Apple, E Pickett, M Palmer, A Ametani, E E Sercarz

J Exp Med (1991) 173 (3): 609–617.

DOI: https://doi.org/10.1084/jem.173.3.609

Published: 01 March 1991

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Journal Articles

Immunogenicity and tolerogenicity of self-major histocompatibility complex peptides.

G Benichou, P A Takizawa, P T Ho, C C Killion, C A Olson, M McMillan, E E Sercarz

J Exp Med (1990) 172 (5): 1341–1346.

DOI: https://doi.org/10.1084/jem.172.5.1341

Published: 01 November 1990

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Journal Articles

Peptide-specific prevention of experimental allergic encephalomyelitis. Neonatal tolerance induced to the dominant T cell determinant of myelin basic protein.

J P Clayton, G M Gammon, D G Ando, D H Kono, L Hood, E E Sercarz

J Exp Med (1989) 169 (5): 1681–1691.

DOI: https://doi.org/10.1084/jem.169.5.1681

Published: 01 May 1989

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Journal Articles

Ia molecule-associated selectivity in T cell recognition of a 23-amino-acid peptide of lysozyme.

N Shastri, G Gammon, A Miller, E E Sercarz

J Exp Med (1986) 164 (3): 882–896.

DOI: https://doi.org/10.1084/jem.164.3.882

Published: 01 September 1986

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Journal Articles

Induction of tolerance to one determinant on a synthetic peptide does not affect the response to a second linked determinant. Implications for the mechanism of neonatal tolerance induction.

G M Gammon, A Oki, N Shastri, E E Sercarz

J Exp Med (1986) 164 (2): 667–672.

DOI: https://doi.org/10.1084/jem.164.2.667

Published: 01 August 1986

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Journal Articles

Repertoires of T cells directed against a large protein antigen, beta-galactosidase. II. Only certain T helper or T suppressor cells are relevant in particular regulatory interactions.

U Krzych, A V Fowler, E E Sercarz

J Exp Med (1985) 162 (1): 311–323.

DOI: https://doi.org/10.1084/jem.162.1.311

Published: 01 July 1985

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NARROW

T Cell Receptor Complementarity Determining Region 3 Length Analysis Reveals the Absence of a Characteristic Public T Cell Repertoire in Neonatal Tolerance: The Response in the “Tolerant” Mouse within the Residual Repertoire Is Quantitatively Similar but Qualitatively Different

Diversification of T Cell Responses to Carboxy-terminal Determinants within the 65-kD Heat-shock Protein Is Involved in Regulation of Autoimmune Arthritis

Inactivation of T cell receptor peptide-specific CD4 regulatory T cells induces chronic experimental autoimmune encephalomyelitis (EAE).

A truncated T cell receptor repertoire reveals underlying immunogenicity of an antigenic determinant.

Neonatal peptide exposure can prime T cells and, upon subsequent immunization, induce their immune deviation: implications for antibody vs. T cell-mediated autoimmunity.

Unresponsiveness to a self-peptide of mouse lysozyme owing to hindrance of T cell receptor-major histocompatibility complex/peptide interaction caused by flanking epitopic residues.

Recognition of multiple peptide cores by a single T cell receptor.

T cell determinants from autoantibodies to DNA can upregulate autoimmunity in murine systemic lupus erythematosus.

Holes in the T cell repertoire to myelin basic protein owing to the absence of the D beta 2-J beta 2 gene cluster: implications for T cell receptor recognition and autoimmunity.

Dominant determinants in hen eggwhite lysozyme correspond to the cryptic determinants within its self-homologue, mouse lysozyme: implications in shaping of the T cell repertoire and autoimmunity.

Determinant capture as a possible mechanism of protection afforded by major histocompatibility complex class II molecules in autoimmune disease.

The involvement of T cell receptor peptide-specific regulatory CD4+ T cells in recovery from antigen-induced autoimmune disease.

In a small multideterminant peptide, each determinant is recognized by a different V beta gene segment.

Donor major histocompatibility complex (MHC) peptides are presented by recipient MHC molecules during graft rejection.

T cell determinant structure: cores and determinant envelopes in three mouse major histocompatibility complex haplotypes.

Immunogenicity and tolerogenicity of self-major histocompatibility complex peptides.

Peptide-specific prevention of experimental allergic encephalomyelitis. Neonatal tolerance induced to the dominant T cell determinant of myelin basic protein.

Ia molecule-associated selectivity in T cell recognition of a 23-amino-acid peptide of lysozyme.

Induction of tolerance to one determinant on a synthetic peptide does not affect the response to a second linked determinant. Implications for the mechanism of neonatal tolerance induction.

Repertoires of T cells directed against a large protein antigen, beta-galactosidase. II. Only certain T helper or T suppressor cells are relevant in particular regulatory interactions.

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