Page 1 | Search Results | Journal of Experimental Medicine

Skip Nav Destination

1-6 of 6

Sort by

Sort Order Select

Journal Articles

11β-HSD1 inhibition ameliorates metabolic syndrome and prevents progression of atherosclerosis in mice

J Exp Med (2005) 202 (4): 517–527.

https://doi.org/10.1084/jem.20050119

Published: 15 August 2005

View article titled, 11β-HSD1 inhibition ameliorates metabolic syndrome and prevents progression of atherosclerosis in mice

Journal Articles

Infectious Agents Are Not Necessary for Murine Atherogenesis

Samuel D. Wright, Charlotte Burton, Melba Hernandez, Heide Hassing, Judy Montenegro, Steve Mundt, Sushma Patel, Deborah J. Card, Anne Hermanowski-Vosatka, James D. Bergstrom, Carl P. Sparrow, Patricia A. Detmers, Yu-Sheng Chao

J Exp Med (2000) 191 (8): 1437–1442.

https://doi.org/10.1084/jem.191.8.1437

Published: 18 April 2000

View article titled, Infectious Agents Are Not Necessary for Murine Atherogenesis

Journal Articles

Transport of Bacterial Lipopolysaccharide to the Golgi Apparatus

Nathalie Thieblemont, Samuel D. Wright

J Exp Med (1999) 190 (4): 523–534.

https://doi.org/10.1084/jem.190.4.523

Published: 16 August 1999

View article titled, Transport of Bacterial Lipopolysaccharide to the Golgi Apparatus

Journal Articles

Toll, A New Piece in the Puzzle of Innate Immunity

Samuel D. Wright

J Exp Med (1999) 189 (4): 605–609.

https://doi.org/10.1084/jem.189.4.605

Published: 15 February 1999

View article titled, Toll, A New Piece in the Puzzle of Innate Immunity

Journal Articles

Targeted Deletion of the Lipopolysaccharide (LPS)-binding Protein Gene Leads to Profound Suppression of LPS Responses Ex Vivo, whereas In Vivo Responses Remain Intact

Mark M. Wurfel, Brian G. Monks, Robin R. Ingalls, Russell L. Dedrick, Russell Delude, Dahua Zhou, Norbert Lamping, Ralf R. Schumann, Rolf Thieringer, Matthew J. Fenton, Samuel D. Wright, Douglas Golenbock

J Exp Med (1997) 186 (12): 2051–2056.

https://doi.org/10.1084/jem.186.12.2051

Published: 15 December 1997

View article titled, Targeted Deletion of the Lipopolysaccharide (LPS)-binding Protein Gene Leads to Profound Suppression of LPS Responses Ex Vivo, whereas In Vivo Responses Remain Intact

Journal Articles

Mice Genetically Hyporesponsive to Lipopolysaccharide (LPS) Exhibit a Defect in Endocytic Uptake of LPS and Ceramide

Nathalie Thiéblemont, Samuel D. Wright

J Exp Med (1997) 185 (12): 2095–2100.

https://doi.org/10.1084/jem.185.12.2095

Published: 16 June 1997

View article titled, Mice Genetically Hyporesponsive to Lipopolysaccharide (LPS) Exhibit a Defect in Endocytic Uptake of LPS and Ceramide

NARROW

11β-HSD1 inhibition ameliorates metabolic syndrome and prevents progression of atherosclerosis in mice

Infectious Agents Are Not Necessary for Murine Atherogenesis

Transport of Bacterial Lipopolysaccharide to the Golgi Apparatus

Toll, A New Piece in the Puzzle of Innate Immunity

Targeted Deletion of the Lipopolysaccharide (LPS)-binding Protein Gene Leads to Profound Suppression of LPS Responses Ex Vivo, whereas In Vivo Responses Remain Intact

Mice Genetically Hyporesponsive to Lipopolysaccharide (LPS) Exhibit a Defect in Endocytic Uptake of LPS and Ceramide

Email alerts

11β-HSD1 inhibition ameliorates metabolic syndrome and prevents progression of atherosclerosis in mice

Infectious Agents Are Not Necessary for Murine Atherogenesis

Transport of Bacterial Lipopolysaccharide to the Golgi Apparatus

Toll, A New Piece in the Puzzle of Innate Immunity

Targeted Deletion of the Lipopolysaccharide (LPS)-binding Protein Gene Leads to Profound Suppression of LPS Responses Ex Vivo, whereas In Vivo Responses Remain Intact

Mice Genetically Hyporesponsive to Lipopolysaccharide (LPS) Exhibit a Defect in Endocytic Uptake of LPS and Ceramide

Email alerts

This Feature Is Available To Subscribers Only