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Journal Articles

Transgenic expression of the Ly49A natural killer cell receptor confers class I major histocompatibility complex (MHC)-specific inhibition and prevents bone marrow allograft rejection.

W Held, D Cado, D H Raulet

J Exp Med (1996) 184 (5): 2037–2041.

https://doi.org/10.1084/jem.184.5.2037

Published: 01 November 1996

View article titled, Transgenic expression of the Ly49A natural killer cell receptor confers class I major histocompatibility complex (MHC)-specific inhibition and prevents bone marrow allograft rejection.

Journal Articles

Reverse transcriptase-dependent and -independent phases of infection with mouse mammary tumor virus: implications for superantigen function.

W Held, G A Waanders, H Acha-Orbea, H R MacDonald

J Exp Med (1994) 180 (6): 2347–2351.

https://doi.org/10.1084/jem.180.6.2347

Published: 01 December 1994

View article titled, Reverse transcriptase-dependent and -independent phases of infection with mouse mammary tumor virus: implications for superantigen function.

Journal Articles

Two waves of recombinase gene expression in developing thymocytes.

A Wilson, W Held, H R MacDonald

J Exp Med (1994) 179 (4): 1355–1360.

https://doi.org/10.1084/jem.179.4.1355

Published: 01 April 1994

View article titled, Two waves of recombinase gene expression in developing thymocytes.

Journal Articles

Peripheral T cell activation and deletion induced by transfer of lymphocyte subsets expressing endogenous or exogenous mouse mammary tumor virus.

G A Waanders, A N Shakhov, W Held, O Karapetian, H Acha-Orbea, H R MacDonald

J Exp Med (1993) 177 (5): 1359–1366.

https://doi.org/10.1084/jem.177.5.1359

Published: 01 May 1993

View article titled, Peripheral T cell activation and deletion induced by transfer of lymphocyte subsets expressing endogenous or exogenous mouse mammary tumor virus.

Journal Articles

Superantigen-reactive CD4+ T cells are required to stimulate B cells after infection with mouse mammary tumor virus.

W Held, A N Shakhov, S Izui, G A Waanders, L Scarpellino, H R MacDonald, H Acha-Orbea

J Exp Med (1993) 177 (2): 359–366.

https://doi.org/10.1084/jem.177.2.359

Published: 01 February 1993

View article titled, Superantigen-reactive CD4+ T cells are required to stimulate B cells after infection with mouse mammary tumor virus.

Journal Articles

Inhibition of mouse mammary tumor virus-induced T cell responses in vivo by antibodies to an open reading frame protein.

H Acha-Orbea, L Scarpellino, A N Shakhov, W Held, H R MacDonald

J Exp Med (1992) 176 (6): 1769–1772.

https://doi.org/10.1084/jem.176.6.1769

Published: 01 December 1992

View article titled, Inhibition of mouse mammary tumor virus-induced T cell responses in vivo by antibodies to an open reading frame protein.

Journal Articles

An exogenous mouse mammary tumor virus with properties of Mls-1a (Mtv-7).

W Held, A N Shakhov, G Waanders, L Scarpellino, R Luethy, J P Kraehenbuhl, H R MacDonald, H Acha-Orbea

J Exp Med (1992) 175 (6): 1623–1633.

https://doi.org/10.1084/jem.175.6.1623

Published: 01 June 1992

View article titled, An exogenous mouse mammary tumor virus with properties of Mls-1a (Mtv-7).

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Transgenic expression of the Ly49A natural killer cell receptor confers class I major histocompatibility complex (MHC)-specific inhibition and prevents bone marrow allograft rejection.

Reverse transcriptase-dependent and -independent phases of infection with mouse mammary tumor virus: implications for superantigen function.

Two waves of recombinase gene expression in developing thymocytes.

Peripheral T cell activation and deletion induced by transfer of lymphocyte subsets expressing endogenous or exogenous mouse mammary tumor virus.

Superantigen-reactive CD4+ T cells are required to stimulate B cells after infection with mouse mammary tumor virus.

Inhibition of mouse mammary tumor virus-induced T cell responses in vivo by antibodies to an open reading frame protein.

An exogenous mouse mammary tumor virus with properties of Mls-1a (Mtv-7).

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Transgenic expression of the Ly49A natural killer cell receptor confers class I major histocompatibility complex (MHC)-specific inhibition and prevents bone marrow allograft rejection.

Reverse transcriptase-dependent and -independent phases of infection with mouse mammary tumor virus: implications for superantigen function.

Two waves of recombinase gene expression in developing thymocytes.

Peripheral T cell activation and deletion induced by transfer of lymphocyte subsets expressing endogenous or exogenous mouse mammary tumor virus.

Superantigen-reactive CD4+ T cells are required to stimulate B cells after infection with mouse mammary tumor virus.

Inhibition of mouse mammary tumor virus-induced T cell responses in vivo by antibodies to an open reading frame protein.

An exogenous mouse mammary tumor virus with properties of Mls-1a (Mtv-7).

Email alerts

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