Genetic interactions between car-1 and puf-5
| Genotypea . | RNAi . | Small oocyte phenotypeb . | Yolk accumulationc . | Embryo defectsd . | . | |
|---|---|---|---|---|---|---|
. | . | . | . | Egg shell defects . | Cytokinesis defects . | |
| % | % | % | % | |||
| +/+ (N2) | empty vector | 2 (58) | 0 (29) | 1 (104) | 0 (219) | |
| +/hT2 | puf-5 | 0 (28) | 0 (23) | 8 (209) | 8 (209) | |
| car-1/hT2 | empty vector | 0 (55) | 0 (48) | 0 (244) | 1 (357) | |
| car-1/hT2 | puf-5 | 75 (85) | 96 (73) | 91 (232) | 98 (174) | |
| car-1/car-1 | empty vector | 23 (39) | 0 (37) | nd | nd | |
| car-1/car-1 | puf-5 | 86 (36) | 84 (37) | 90 (49) | 98 (203) | |
| +/+ (N2) | puf-5 | 3 (87) | 0 (51) | 4 (246) | 4 (234) | |
| +/+ (N2) | car-1 | 22 (9) | 0 (9) | 2 (80) | 88 (80) | |
| +/+ (N2) | puf-5;car-1 | 75 (8) | 100 (8) | 97 (70) | 97 (70) | |
| Genotypea . | RNAi . | Small oocyte phenotypeb . | Yolk accumulationc . | Embryo defectsd . | . | |
|---|---|---|---|---|---|---|
. | . | . | . | Egg shell defects . | Cytokinesis defects . | |
| % | % | % | % | |||
| +/+ (N2) | empty vector | 2 (58) | 0 (29) | 1 (104) | 0 (219) | |
| +/hT2 | puf-5 | 0 (28) | 0 (23) | 8 (209) | 8 (209) | |
| car-1/hT2 | empty vector | 0 (55) | 0 (48) | 0 (244) | 1 (357) | |
| car-1/hT2 | puf-5 | 75 (85) | 96 (73) | 91 (232) | 98 (174) | |
| car-1/car-1 | empty vector | 23 (39) | 0 (37) | nd | nd | |
| car-1/car-1 | puf-5 | 86 (36) | 84 (37) | 90 (49) | 98 (203) | |
| +/+ (N2) | puf-5 | 3 (87) | 0 (51) | 4 (246) | 4 (234) | |
| +/+ (N2) | car-1 | 22 (9) | 0 (9) | 2 (80) | 88 (80) | |
| +/+ (N2) | puf-5;car-1 | 75 (8) | 100 (8) | 97 (70) | 97 (70) | |
Numbers in parentheses indicate the number of gonads,b animals,c or embryosd scored. nd, not determined.
“car-1” is the car-1(tm1753) deletion allele; hT2 is a balancer chromosome carrying car-1(+).
Percentage of gonads in which >1 proximal oocytes did not span the dorsal/ventral gonad borders.
Percentage of animals with large yolk droplets in the pseudoceloem.
Percentage of embryos with undetectable egg shells or cytokinesis defects as in Fig. 7 F.